Difference between revisions of "Takotsubo cardiomyopathy associated with status epilepticus"

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(Created page with "''Seow SC, Lee YP, Teo SG, Hong ECT, and Lee CH (2008) Takotsubo cardiomyopathy associated with status epilepticus. Eur J Neurol p. e46.'' '''[http://onlinelibrary.wiley.com/...")
 
 
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''Seow SC, Lee YP, Teo SG, Hong ECT, and Lee CH (2008) Takotsubo cardiomyopathy associated with status epilepticus. Eur J Neurol p. e46.''
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'''[http://onlinelibrary.wiley.com/doi/10.1111/j.1468-1331.2008.02125.x/epdf Link to Article]'''
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Seow SC, Lee YP, Teo SG, Hong ECT, and Lee CH (2008) Takotsubo cardiomyopathy associated with status epilepticus. Eur J Neurol p. e46.
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http://onlinelibrary.wiley.com/doi/10.1111/j.1468-1331.2008.02125.x/epdf
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'''First Paragraph:''' A 62-year-old Chinese male with no previous history of seizures was admitted with status epilepticus necessitating intravenous lorazepam, propofol and endotracheal intubation. He became hypotensive and an ECG showed ST-segment elevation in the anterior precordial leads. Cardiac enzymes were elevated. He did not have any cardiovascular risk factors. Bedside echocardiogram showed mildly impaired left ventricular ejection fraction of 40% with mid-ventricular ballooning and relative sparing of the apex. Coronary angiogram revealed a non-significant stenosis in the obtuse marginal branch of the left circumflex artery. A diagnosis of Takotsubo (stress-induced) cardiomyopathy was made. The patient subsequently made an uneventful cardiac recovery. Repeat echocardiogram 6 weeks later was normal. MRI of the brain showed encephalomalacia of the right basal frontal lobe with old lacunas in bilateral external capsules, bilateral corona radiata and right hemipons.
 
'''First Paragraph:''' A 62-year-old Chinese male with no previous history of seizures was admitted with status epilepticus necessitating intravenous lorazepam, propofol and endotracheal intubation. He became hypotensive and an ECG showed ST-segment elevation in the anterior precordial leads. Cardiac enzymes were elevated. He did not have any cardiovascular risk factors. Bedside echocardiogram showed mildly impaired left ventricular ejection fraction of 40% with mid-ventricular ballooning and relative sparing of the apex. Coronary angiogram revealed a non-significant stenosis in the obtuse marginal branch of the left circumflex artery. A diagnosis of Takotsubo (stress-induced) cardiomyopathy was made. The patient subsequently made an uneventful cardiac recovery. Repeat echocardiogram 6 weeks later was normal. MRI of the brain showed encephalomalacia of the right basal frontal lobe with old lacunas in bilateral external capsules, bilateral corona radiata and right hemipons.
  
'''Keywords:''' apical ballooning, status epilepticus, Takotsubo cardiomyopathy
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apical ballooning, status epilepticus, Takotsubo cardiomyopathy
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*Letter to the editor single-case report of Takotsubo cardiomyopathy – midsystolic apical ballooning – detected on echocardiography in a patient who had developed EKG changes and hypotension during status epilepticus. The authors state that this acute cardiomyopathy is “thought to be due to catecholamine-induced coronary microcirculatory spasm,” suggesting that in this patient with no cardiovascular risk factors the onset of status epilepticus may have produced a significant increase in circulatory catecholamines.
 
*Letter to the editor single-case report of Takotsubo cardiomyopathy – midsystolic apical ballooning – detected on echocardiography in a patient who had developed EKG changes and hypotension during status epilepticus. The authors state that this acute cardiomyopathy is “thought to be due to catecholamine-induced coronary microcirculatory spasm,” suggesting that in this patient with no cardiovascular risk factors the onset of status epilepticus may have produced a significant increase in circulatory catecholamines.
  
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Latest revision as of 14:07, 17 June 2019


Seow SC, Lee YP, Teo SG, Hong ECT, and Lee CH (2008) Takotsubo cardiomyopathy associated with status epilepticus. Eur J Neurol p. e46.

Link to Article

Abstract: First Paragraph: A 62-year-old Chinese male with no previous history of seizures was admitted with status epilepticus necessitating intravenous lorazepam, propofol and endotracheal intubation. He became hypotensive and an ECG showed ST-segment elevation in the anterior precordial leads. Cardiac enzymes were elevated. He did not have any cardiovascular risk factors. Bedside echocardiogram showed mildly impaired left ventricular ejection fraction of 40% with mid-ventricular ballooning and relative sparing of the apex. Coronary angiogram revealed a non-significant stenosis in the obtuse marginal branch of the left circumflex artery. A diagnosis of Takotsubo (stress-induced) cardiomyopathy was made. The patient subsequently made an uneventful cardiac recovery. Repeat echocardiogram 6 weeks later was normal. MRI of the brain showed encephalomalacia of the right basal frontal lobe with old lacunas in bilateral external capsules, bilateral corona radiata and right hemipons.

Keywords: apical ballooning, status epilepticus, Takotsubo cardiomyopathy

Context

  • Letter to the editor single-case report of Takotsubo cardiomyopathy – midsystolic apical ballooning – detected on echocardiography in a patient who had developed EKG changes and hypotension during status epilepticus. The authors state that this acute cardiomyopathy is “thought to be due to catecholamine-induced coronary microcirculatory spasm,” suggesting that in this patient with no cardiovascular risk factors the onset of status epilepticus may have produced a significant increase in circulatory catecholamines.

Comments

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